Publications

2018


Weavers, H., Franz, A., Wood, W., & Martin, P. (2018). Long-term In Vivo Tracking of Inflammatory Cell Dynamics Within Drosophila Pupae. doi:10.3791/57871





 

Gordon, O., Henry, C. M., Srinivasan, N., Ahrens, S., Franz, A., Deddouche, S., . . . Reis E Sousa, C. (2018). α-actinin accounts for the bioactivity of actin preparations in inducing STAT target genes in Drosophila melanogaster.  e38636. doi:10.7554/eLife.38636



Franz, A., Wood, W., & Martin, P. (2018). Fat Body Cells Are Motile and Actively Migrate to Wounds to Drive Repair and Prevent Infection. 460-470.e3. doi:10.1016/j.devcel.2018.01.026




Previewed in Dev Cell 2018, Casano AM and Sixt M “A Fat Lot of Good for Wound Healing”, featured in the New York Times article: “Inside Wounded Flies, Fat Cells Race to the Rescue” and F1000Prime recommended.






2016

Srinivasan, N., Gordon, O., Ahrens, S., Franz, A., Deddouche, S., Chakravarty, P., . . . Reis e Sousa, C. (2016). Actin is an evolutionarily-conserved damage-associated molecular pattern that signals tissue injury in Drosophila melanogaster. doi:10.7554/eLife.19662






 

2013

Franz, A., Roque, H., Saurya, S., Dobbelaere, J., & Raff, J. W. (2013). CP110 exhibits novel regulatory activities during centriole assembly in Drosophila. 785-799. doi:10.1083/jcb.201305109






 

2012

Roque, H., Wainman, A., Richens, J., Kozyrska, K., Franz, A., & Raff, J. W. (2012). Drosophila Cep135/Bld10 maintains proper centriole structure but is dispensable for cartwheel formation. 5881-5886. doi:10.1242/jcs.113506








2010

Stevens, N. R., Dobbelaere, J., Brunk, K., Franz, A., & Raff, J. W. (2010). Drosophila Ana2 is a conserved centriole duplication factor. 313-323. doi:10.1083/jcb.200910016






2008

Basto, R., Brunk, K., Vinadogrova, T., Peel, N., Franz, A., Khodjakov, A., & Raff, J. W. (2008). Centrosome amplification can initiate tumorigenesis in flies. 1032-1042. doi:10.1016/j.cell.2008.05.039





2007

Franz, A., Maass, K., & Seedorf, M. (2007). A complex peptide-sorting signal, but no mRNA signal, is required for the Sec-independent transport of Ist2 from the yeast ER to the plasma membrane. 401-405. doi:10.1016/j.febslet.2006.12.048